Human Gut Commensal Membrane Vesicles Modulate Inflammation by Generating M2-like Macrophages and Myeloid-Derived Suppressor Cells.
MouseWoundFemaleIntraperitoneal
Model: Excisional skin wound, alongside acute inflammation models comprising dextran sodium sulfate colitis, zymosan peritonitis and carbon tetrachloride induced fibrosis
Membrane vesicles ameliorated inflammation across the acute models and accelerated excisional wound closure, associated with recruitment of PD-L1 expressing myeloid cells to the wound site.
Extraction: extract · Supplied extraction confidence: 0.6. Machine-extracted fields require source verification.