Sesamin promotes angiogenesis and accelerates wound healing in rats via alleviates TBHP-induced apoptosis in human umbilical vein endothelial cells.
RatWoundSprague-Dawley
Model: Cutaneous wound in rats, with parallel tert-butyl hydroperoxide-induced apoptosis in human umbilical vein endothelial cells
Sesamin dose-dependently increased endothelial proliferation, adherence, migration and angiogenic capacity, attenuated tert-butyl hydroperoxide-induced apoptosis, and accelerated granulation tissue formation and collagen deposition and remodelling in rat wounds.
Extraction: extract · Supplied extraction confidence: 0.5. Machine-extracted fields require source verification.