Attenuation of kidney injuries maintains serum sulfatide levels dependent on hepatic synthetic ability: a possible involvement of oxidative stress.

Attenuation of kidney injuries maintains serum sulfatide levels dependent on hepatic synthetic ability: a possible involvement of oxidative stress.

Model: Protein-overload nephropathy with acute kidney injury induced by bovine serum albumin loading

Clofibrate attenuated kidney damage and hepatic oxidative stress while maintaining serum and hepatic sulfatide levels and hepatic cerebroside sulfotransferase content. Clofibrate alone without protein overload had only minimal effects on these hepatic parameters.

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